herpes
 

Key to Herpes Infection Could Lead to Effective Drugs

by John Stuttmier

Researchers at Tufts University have discovered a unique structure of the protein complex that allows Herpes virus to infect new cells. This information opens up a new strategy that new anti-viral herpes drugs could use to prevent spread of herpes.

Co-author of the reseach findings published in Nature Structural & Molecular Biology and assistant professor in the molecular biology and microbiology department at Tufts University School of Medicine, Ekaterina Heldwein, PhD, states "Most viruses need cell-entry proteins called fusogens in order to invade cells. We have known that the herpes virus fusogen does not act alone and that a complex of two other viral cell-entry proteins is always required. We expected that this complex was also a fusogen, but after determining the structure of this key protein complex, we found that it does not resemble other known fusogens,"

"This unexpected result leads us to believe that this protein complex is not a fusogen itself but that it regulates the fusogen. We also found that certain antibodies interfere with the ability of this protein complex to bind to the fusogen, evidence that antiviral drugs that target this interaction could prevent viral infection," Heldwein added.

This type of research breaks open many new possibilities for effective herpes medications in the near future. These new medications could not only stop outbreaks, but also prevent the spread of HSV to uninfected partners.

You can read more about this promising break-through:
Map of Herpes Virus Protein Suggests a New Drug Therapy

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